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《循环研究杂志》2005年第04期

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atherosclerotic abdominal aortic aneurysm and the interaction between autologous human plaque-derived vascular smooth muscle cells, type 1 nkt, and helper t cells
platelets and chemokines in atherosclerosis
a role for endoglin in coupling enos activity and regulating vascular tone revealed in hereditary hemorrhagic telangiectasia
common variants in myocardial ion channel genes modify the qt interval in the general population
activation of inflammation and coagulation after infusion of c-reactive protein in humans
regulation of vascular calcification
role of antioxidant-1 in extracellular superoxide dismutase function and expression
impaired kcnq1eckcne1 and phosphatidylinositol-4,5-bisphosphate interaction underlies the long qt syndrome
functional effects of rho-kinaseecdependent phosphorylation of specific sites on cardiac troponin
increased collagen deposition and diastolic dysfunction but preserved myocardial hypertrophy after pressure overload in mice lacking pkc
enhancement of cardiac function and suppression of heart failure progression by inhibition of protein phosphatase 1
adenoviral gene transfer of fgf-5 to hibernating myocardium improves function and stimulates myocytes to hypertrophy and reenter the cell cycle
identification of hypertension-related genes through an integrated genomic-transcriptomic approach
development of the right ventricular inflow tract and moderator band
sdf-1/cxcr4 axis is instrumental in neointimal hyperplasia and recruitment of smooth muscle progenitor cells
spatial heterogeneity of endothelial phenotypes correlates with side-specific vulnerability to calcification in normal porcine aortic valves
a novel form of short qt syndrome (sqt3) is caused by a mutation in the kcnj2 gene
liver x receptor agonists inhibit cytokine-induced osteopontin expression in macrophages through interference with activator protein-1 signaling pathways
adenoviral gene transfer of mutant phospholamban rescues contractile dysfunction in failing rabbit myocytes with relatively preserved serca function
nad(p)h oxidaseecdependent self-propagation of hydrogen peroxide and vascular disease
hepatocyte growth factor ameliorates the progression of experimental autoimmune myocarditis
adenosine-dependent induction of glutathione peroxidase 1 in human primary endothelial cells and protection against oxidative stress
amp-activated protein kinase signaling stimulates vegf expression and angiogenesis in skeletal muscle
distinct nf-b regulation by shear stress through ras-dependent ib oscillations
characterization of a novel pka phosphorylation site, serine-2030, reveals no pka hyperphosphorylation of the cardiac ryanodine receptor in canine heart failure
essential role of a ca2+-selective, store-operated current (isoc) in endothelial cell permeability
low-voltage-activated (t-type) calcium channels control proliferation of human pulmonary artery myocytes
[ca2+]i reduction increases cellular proliferation and apoptosis in vascular smooth muscle cells
ccl2/monocyte chemoattractant protein-1 regulates inflammatory responses critical to healing myocardial infarcts
contractile properties of the cultured vascular smooth muscle cells
homeobox protein hop functions in the adult cardiac conduction system
targeted disruption of tgf-ecsmad3 signaling leads to enhanced neointimal hyperplasia with diminished matrix deposition in response to vascular injury
immunomodulator fty720 induces enos-dependent arterial vasodilatation via the lysophospholipid receptor s1p3
lack of myoglobin causes a switch in cardiac substrate selection
caveolin-1 facilitates mechanosensitive protein kinase b (akt) signaling in vitro and in vivo
angiotensin ii and aldosterone regulate gene transcription via functional mineralocortocoid receptors in human coronary artery smooth muscle cells
triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes
nad(p)h oxidase inhibition attenuates neuronal chronotropic actions of angiotensin ii
endogenous p53 protects vascular smooth muscle cells from apoptosis and reduces atherosclerosis in apoe knockout mice
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