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《细胞学杂志》2005年第17期

lis-less neurons fall behind
formin steps and slips
a notch outlier
keeping rna away from dna
an actin ruler
getting nuclei moving
porterplasm" and the microtrabecular lattice
turning on by turning
the isolation of the nuclear lamina
freedom not aggregation
the eif2 translation initiation factor travels to a cytoplasmic focus for activation, according to campbell et al. (page 925). in stressed cells, which turn down tr..
an optical brain
defective fas expression exacerbates neurotoxicity in a model of parkinson's disease
lis1 rna interference blocks neural stem cell division, morphogenesis, and motility at multiple stages
lis-less neurons don't even make it to the starting gate
syntabulin-mediated anterograde transport of mitochondria along neuronal processes
external scaffold of spherical immature poxvirus particles is made of protein trimers, forming a honeycomb lattice
a novel mechanism of actin filament processive capping by formin : solution of the rotation paradox
nebulin regulates the assembly and lengths of the thin filaments in striated muscle
microtubule capture by cenp-e silences bubr1-dependent mitotic checkpoint signaling
cdc42 and par6
dynamic cycling of eif2 through a large eif2b-containing cytoplasmic body : implications for translation control
proline residues of transmembrane domains determine the sorting of inner membrane proteins in mitochondria
tourriere, h., k. cha role for the eif4e-binding protein 4e-t in p-body formation and mrna decayebli, and j. tazi. 2002. mrna degradation machines in eukaryotic cel..
stable interaction between 5?1 integrin and tie2 tyrosine kinase receptor regulates endothelial cell response to ang-1
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