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《细胞学杂志》2004年第04期

how meals become bones
a primordial mover for sphingolipids
capping protein binding to actin in yeast : biochemical mechanism and physiological relevance
boning up on junb
yeast dies for a noble cause
protein splicing for immunity
apoptotic mitochondria blow a fuse
nudel functions in membrane traffic mainly through association with lis1 and cytoplasmic dynein
inflexibility in motion
breathe, then breed
winding and unwinding atp synthase
nanofibers have the right stuff
cells on parade : the american society for cell biology san francisco, ca december 13
greatwall kinase : a nuclear protein required for proper chromosome condensation and mitotic progression in drosophila
quantitation of mitochondrial dynamics by photolabeling of individual organelles shows that mitochondrial fusion is blocked during the bax activation phase of apopt..
amylin inhibits bone resorption while the calcitonin receptor controls bone formation in vivo
chronological aging leads to apoptosis in yeast
promyelocytic leukemia nuclear bodies associate with transcriptionally active genomic regions
bicarbonate-responsive "soluble" adenylyl cyclase defines a nuclear camp microdomain
mice lacking junb are osteopenic due to cell-autonomous osteoblast and osteoclast defects
in candida albicans, the nim1 kinases gin4 and hsl1 negatively regulate pseudohypha formation and gin4 also controls septin organization
phosphorylation of paxillin by p38mapk is involved in the neurite extension of pc-12 cells
polarity and proliferation are controlled by distinct signaling pathways downstream of pi3-kinase in breast epithelial tumor cells
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