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《细胞生物学杂志》2006年第06期

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identification of a novel peptide that interferes with the chemical regulation of connexin43
common protective and diverse smooth muscle cell effects of aav-mediated angiopoietin-1 and -2 expression in rat cardiac allograft vasculopathy
a sphingosine-1
insulin-like growth factor-1 and pten deletion enhance cardiac l-type ca2+ currents via increased pi3k/pkb signaling
syndecan-4 clustering induces cell migration in a pdz-dependent manner
erythropoietin-mobilized endothelial progenitors enhance reendothelialization via akt-endothelial nitric oxide synthase activation and prevent neointimal hyperplasia
bone marrow stem cells prevent left ventricular remodeling of ischemic heart through paracrine signaling
bradycardia and slowing of the atrioventricular conduction in mice lacking cav3.1/1g t-type calcium channels
extracellular matrix remodeling and organization in developing and diseased aortic valves
increased expression of cyclooxygenase-2 mediates enhanced contraction to endothelin eta receptor stimulation in endothelial nitric oxide synthase knockout mice
axl, a receptor tyrosine kinase, mediates flow-induced vascular remodeling
induction of cardiogenesis in embryonic stem cells via downregulation of notch1 signaling
an osteopontin
integrin-mediated transcriptional activation of inhibitor of apoptosis proteins protects smooth muscle cells against apoptosis induced by degraded collagen
selectivity of connexin 43 channels is regulated through protein kinase c
maximum phosphorylation of the cardiac ryanodine receptor at serine-2809 by protein kinase a produces unique modifications to channel gating and conductance not obs..
effects of congestive heart failure on ca2+ handling in skeletal muscle during fatigue
heteromultimeric trpc6-trpc7 channels contribute to arginine vasopressin-induced cation current of a7r5 vascular smooth muscle cells
hypoxia inducible factor 1 mediates hypoxia-induced trpc expression and elevated intracellular ca2+ in pulmonary arterial smooth muscle cells
14-3-3 is a regulator of the cardiac voltage-gated sodium channel nav1.5
neural crest cells retain multipotential characteristics in the developing valves and label the cardiac conduction system
formation of the venous pole of the heart from an nkx2
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