互联网 qkzz.net
全刊杂志网:首页 > 医药医学 > 《糖尿病学杂志》 > 2006年第06期
《糖尿病学杂志》
糖尿病学杂志2006年第06期封面 购买
收藏
投稿

《糖尿病学杂志》2006年第06期

[文章精华]
inhibition of fructose 1,6-bisphosphatase reduces excessive endogenous glucose production and attenuates hyperglycemia in zucker diabetic fatty rats
induction of tolerance in type 1 diabetes via both cd4+cd25+ t regulatory cells and t regulatory type 1 cells
glucose regulates foxo1 through insulin receptor signaling in the pancreatic islet -cell
metallothionein and catalase sensitize to diabetes in nonobese diabetic mice
peroxisome proliferatorecactivated receptor improves pancreatic adaptation to insulin resistance in obese mice and reduces lipotoxicity in human islets
the role of phosphoinositide 3-kinase/akt signaling in low-dose mercuryecinduced mouse pancreatic -cell dysfunction in vitro and in vivo
islet inflammation and fibrosis in a spontaneous model of type 2 diabetes, the gk rat
nutritional supplementation with trans-10, cis-12ecconjugated linoleic acid induces inflammation of white adipose tissue
cognitive performance, psychological well-being, and brain magnetic resonance imaging in older patients with type 1 diabetes
hyperglycemia stimulates coagulation, whereas hyperinsulinemia impairs fibrinolysis in healthy humans
decreased muscle strength and quality in older adults with type 2 diabetes
lipid storage compartment to endocrine organ
the intrauterine environment as reflected by birth size and twin and zygosity status influences insulin action and intracellular glucose metabolism in an age- or ti..
deletion of p66shc longevity gene protects against experimental diabetic glomerulopathy by preventing diabetes-induced oxidative stress
altered endothelial nitric oxide synthase targeting and conformation and caveolin-1 expression in the diabetic kidney
apoptosis signal-regulating kinase 1 mediates cellular senescence induced by high glucose in endothelial cells
thiazolidinediones ameliorate diabetic nephropathy via cell cycleecdependent mechanisms
salutary effect of pigment epitheliumecderived factor in diabetic nephropathy
poly(adp-ribose) polymerase inhibition alleviates experimental diabetic sensory neuropathy
diabetic nephropathy is associated with gene expression levels of oxidative phosphorylation and related pathways
risk factors for renal dysfunction in type 2 diabetes
adiponectin suppression of high-glucoseecinduced reactive oxygen species in vascular endothelial cells
reversal of type 1 diabetes by engineering a glucose sensor in skeletal muscle
gene transfer of an engineered transcription factor promoting expression of vegf-a protects against experimental diabetic neuropathy
chronic inhibition of dipeptidyl peptidase-4 with a sitagliptin analog preserves pancreatic -cell mass and function in a rodent model of type 2 diabetes
mutations at the same residue (r50) of kir6.2 (kcnj11) that cause neonatal diabetes produce different functional effects
clinicogenetic studies of maturity-onset diabetes of the young to unraveling complex mechanisms of glucokinase regulation
genome-wide linkage of plasma adiponectin reveals a major locus on chromosome 3q distinct from the adiponectin structural gene
functional effects of mutations at f35 in the nh2-terminus of kir6.2 (kcnj11), causing neonatal diabetes, and response to sulfonylurea therapy
kir6.2 mutations associated with neonatal diabetes reduce expression of atp-sensitive k+ channels
combination of hla-a24, -dqa103, and -dr9 contributes to acute-onset and early complete -cell destruction in type 1 diabetes
exercise-induced phosphorylation of the novel akt substrates as160 and filamin a in human skeletal muscle
evidence for a mitochondrial regulatory pathway defined by peroxisome proliferatorecactivated receptor- coactivator-1, estrogen-related receptor-, and mitofusin 2
activation of amp-activated protein kinase within the ventromedial hypothalamus amplifies counterregulatory hormone responses in rats with defective counterregulation
the experimental type 2 diabetes therapy glycogen phosphorylase inhibition can impair aerobic muscle function during prolonged contraction
a novel ec192c/g mutation in the proximal p2 promoter of the hepatocyte nuclear factor-4 gene (hnf4a) associates with late-onset diabetes
fasting insulin and obesity-related phenotypes are linked to chromosome 2p
linkage but not association of calpain-10 to type 2 diabetes replicated in northern sweden
significant linkage of bmi to chromosome 10p in the u.k. population and evaluation of gad2 as a positional candidate
the variable number of tandem repeats upstream of the insulin gene is a susceptibility locus for latent autoimmune diabetes in adults
hla genotyping supports a nonautoimmune etiology in patients diagnosed with diabetes under the age of 6 months
a hepatocyte nuclear factor-4 gene (hnf4a) p2 promoter haplotype linked with late-onset diabetes
time-dependent effects of free fatty acids on glucose effectiveness in type 2 diabetes
reduced access to insulin-sensitive tissues in dogs with obesity secondary to increased fat intake
increased infiltration of macrophages in omental adipose tissue is associated with marked hepatic lesions in morbid human obesity
downregulation of electron transport chain genes in visceral adipose tissue in type 2 diabetes independent of obesity and possibly involving tumor necrosis factor-
lymphocytic infiltration and immune activation in metallothionein promoterecexendin-4 (mt-exendin) transgenic mice
购买 收藏 投稿
往期回顾
2007年第11期
2007年第10期
2007年第09期
2007年第08期
2007年第07期
2007年第06期
2007年第05期
2007年第04期
2007年第03期
2007年第02期
2007年第01期
2006年第12期
关键字
支持中国杂志产业发展,请购买、订阅纸质杂志,欢迎杂志社提供过刊、样刊及电子版。
关于我们 | 网站声明 | 刊社管理 | 网站地图 | 联系方式 | 中图分类法 | RSS 2.0订阅 | IP查询
全刊杂志赏析网 2017