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《糖尿病学杂志》2005年第11期

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diabetes and insulin secretion
inositol (1,4,5)-trisphosphate dynamics and intracellular calcium oscillations in pancreatic -cells
mechanisms of glucose-induced secretion of pancreatic-derived factor (pander or fam3b) in pancreatic -cells
restitution of defective glucose-stimulated insulin secretion in diabetic gk rat by acetylcholine uncovers paradoxical stimulatory effect of -cell muscarinic recept..
effects of diabetes on ryanodine receptor ca release channel (ryr2) and ca2+ homeostasis in rat heart
metallothionein-mediated antioxidant defense system and its response to exercise training are impaired in human type 2 diabetes
is there a role for locally produced interleukin-1 in the deleterious effects of high glucose or the type 2 diabetes milieu to human pancreatic islets
homocysteine metabolism in zdf (type 2) diabetic rats
insulin resistance, the metabolic syndrome, and incident cardiovascular events in the framingham offspring study
celiac-superior mesenteric ganglionectomy, but not vagotomy, suppresses the sympathoadrenal response to insulin-induced hypoglycemia
two-hour seven-sample oral glucose tolerance test and meal protocol
the human glomerular podocyte is a novel target for insulin action
glycation and carboxymethyllysine levels in skin collagen predict the risk of future 10-year progression of diabetic retinopathy and nephropathy in the diabetes con..
noninvasive monitoring of diabetes-induced cutaneous nerve fiber loss and hypoalgesia in thy1-yfp transgenic mice
aldose reductase deficiency prevents diabetes-induced blood-retinal barrier breakdown, apoptosis, and glial reactivation in the retina of db/db mice
susceptibility to diabetic nephropathy is related to dicarbonyl and oxidative stress
reduced nitric oxide concentration in the renal cortex of streptozotocin-induced diabetic rats
apoptotic stress is counterbalanced by survival elements preventing programmed cell death of dorsal root ganglions in subacute type 1 diabetic bb/wor rats
large genomic rearrangements in the hepatocyte nuclear factor-1 (tcf2) gene are the most frequent cause of maturity-onset diabetes of the young type 5
selection of tagging single nucleotide polymorphisms and association with body fat, insulin sensitivity, and the metabolic syndrome in a normal female population
assessment of 115 candidate genes for diabetic nephropathy by transmission/disequilibrium test
sequence variation in pparg may underlie differential response to troglitazone
partial gene deletion of heart-type fatty acidecbinding protein limits the severity of dietary-induced insulin resistance
liver markers and development of the metabolic syndrome
effect of a sustained reduction in plasma free fatty acid concentration on intramuscular long-chain fatty acyl-coas and insulin action in type 2 diabetic patients
the stimulatory effect of globular adiponectin on insulin-stimulated glucose uptake and fatty acid oxidation is impaired in skeletal muscle from obese subjects
insulin-stimulated glucose uptake does not require p38 mitogen-activated protein kinase in adipose tissue or skeletal muscle
activation of atp-sensitive k+ channels in the ventromedial hypothalamus amplifies counterregulatory hormone responses to hypoglycemia in normal and recurrently hyp..
impaired generation of reactive oxygen species in leprechaunism through downregulation of nox4
central leptin acutely reverses diet-induced hepatic insulin resistance
adipocyte lipases and defect of lipolysis in human obesity
peptide yy(3ec36) inhibits morning, but not evening, food intake and decreases body weight in rhesus macaques
islet cell hormonal responses to hypoglycemia after human islet transplantation for type 1 diabetes
exposure to farming environments in early life and type 1 diabetes
the fatty acidecbinding protein-2 a54t polymorphism is associated with renal disease in patients with type 2 diabetes
risk of diabetic nephropathy in type 1 diabetes is associated with functional polymorphisms in rantes receptor gene (ccr5)
altered calcium homeostasis is correlated with abnormalities of fasting serum glucose, insulin resistance, and -cell function in the newfoundland population
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