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《糖尿病学杂志》2005年第04期

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does an adipokine-induced activation of the immune system mediate the effect of overnutrition on type 2 diabetes
deletion of the angiotensin type 2 receptor (at2r) reduces adipose cell size and protects from diet-induced obesity and insulin resistance
androgen receptor null male mice develop late-onset obesity caused by decreased energy expenditure and lipolytic activity but show normal insulin sensitivity with h..
pdx-1/vp16 fusion protein, together with neurod or ngn3, markedly induces insulin gene transcription and ameliorates glucose tolerance
adipocyte-specific glucocorticoid inactivation protects against diet-induced obesity
reversal of diabetes in pancreatectomized pigs after transplantation of neonatal porcine islets
in vivo control of diabetogenic t-cells by regulatory cd4+cd25+ t-cells expressing foxp3
achieving permanent survival of islet xenografts by independent manipulation of direct and indirect t-cell responses
distinct effects of glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1 on insulin secretion and gut motility
neuronatin, a downstream target of beta2/neurod1 in the pancreas, is involved in glucose-mediated insulin secretion
pancreatic -cell failure and diabetes in mice with a deletion mutation of the endoplasmic reticulum molecular chaperone gene p58ipk
long-term aicar administration and exercise prevents diabetes in zdf rats
inducible nitric oxide synthase has divergent effects on vascular and metabolic function in obesity
serum- and glucocorticoid-inducible kinase 1 (sgk1) mediates glucocorticoid-induced inhibition of insulin secretion
the role of amylin and glucagon in the dampening of glycemic excursions in children with type 1 diabetes
skeletal muscle lipid accumulation in type 2 diabetes may involve the liver x receptor pathway
loss of heparan n-sulfotransferase in diabetic liver
metformin prevents glucose-induced protein kinase c-2 activation in human umbilical vein endothelial cells through an antioxidant mechanism
the calcium-dependent protease calpain causes endothelial dysfunction in type 2 diabetes
nonhuman primate models for diabetic ocular neovascularization using aav2-mediated overexpression of vascular endothelial growth factor
prevention of type 2 diabetes with troglitazone in the diabetes prevention program
direct activation of glucose transport in primary human myotubes after activation of peroxisome proliferatorecactivated receptor
altered hepatic and muscle substrate utilization provoked by glut4 ablation
inhalation of insulin (exubera) is associated with augmented disposal of portally infused glucose in dogs
genetic variations in the gene encoding elmo1 are associated with susceptibility to diabetic nephropathy
the exon 1 cys7gly polymorphism within the betacellulin gene is associated with type 2 diabetes in african americans
genetic analysis of hnf4a polymorphisms in caucasian-american type 2 diabetes
ultrafine mapping of snps from mouse strains c57bl/6j, dba/2j, and c57blks/j for loci contributing to diabetes and atherosclerosis susceptibility
an autosomal genome-wide scan for loci linked to pre-diabetic phenotypes in nondiabetic chinese subjects from the stanford asia-pacific program of hypertension and ..
enpp1/pc-1 k121q polymorphism and genetic susceptibility to type 2 diabetes
a single nucleotide polymorphism in mgea5 encoding o-glcnacecselective n-acetyl--d glucosaminidase is associated with type 2 diabetes in mexican americans
variation in the gene for muscle-specific amp deaminase is associated with insulin clearance, a highly heritable trait
association of the diabetes gene calpain-10 with subclinical atherosclerosis
modular kinetic analysis of the adenine nucleotide translocatorecmediated effects of palmitoyl-coa on the oxidative phosphorylation in isolated rat liver mitochondria
the rat diabetes susceptibility locus iddm4 and at least one additional gene are required for autoimmune diabetes induced by viral infection
genetic variation at the ace gene is associated with persistent microalbuminuria and severe nephropathy in type 1 diabetes
is puberty an accelerator of type 1 diabetes in il6-174cc females
acute induction of gene expression in brain and liver by insulin-induced hypoglycemia
s-nitrosation of the insulin receptor, insulin receptor substrate 1, and protein kinase b/akt
endoplasmic reticulum stressecinduced apoptosis is partly mediated by reduced insulin signaling through phosphatidylinositol 3-kinase/akt and increased glycogen syn..
chronic hyperglycemia enhances pepck gene expression and hepatocellular glucose production via elevated liver activating protein/liver inhibitory protein ratio
insulin, c-peptide, and leptin concentrations predict increased visceral adiposity at 5- and 10-year follow-ups in nondiabetic japanese americans
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