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《糖尿病学杂志》2005年第01期

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perspectives in diabetes
treatment with granulocyte colonyecstimulating factor prevents diabetes in nod mice by recruiting plasmacytoid dendritic cells and functional cd4+cd25+ regulatory t..
hyperglycemia induces monocytic release of interleukin-6 via induction of protein kinase c- and -
defective suppressor function in cd4+cd25+ t-cells from patients with type 1 diabetes
cell function following human islet transplantation for type 1 diabetes
autoimmune diabetes and resistance to xenograft transplantation tolerance in nod mice
uncoupling of nutrient metabolism from insulin secretion by overexpression of cytosolic phospholipase a2
nuclear factor-b activity in -cells is required for glucose-stimulated insulin secretion
genetic background determines the size and structure of the endocrine pancreas
dynamic changes in -cell mass and pancreatic insulin during the evolution of nutrition-dependent diabetes in psammomys obesus
glucagon-like peptide 1 can directly protect the heart against ischemia/reperfusion injury
deficiency of subsarcolemmal mitochondria in obesity and type 2 diabetes
delayed transcapillary delivery of insulin to muscle interstitial fluid after oral glucose load in obese subjects
physical activity in the prevention of type 2 diabetes
predictors of and longitudinal changes in insulin sensitivity and secretion preceding onset of type 2 diabetes
insulin deficiency downregulated heat shock protein 60 and igf-1 receptor signaling in diabetic myocardium
impairment of host resistance to listeria monocytogenes infection in liver of db/db and ob/ob mice
evidence for accelerated rates of glutathione utilization and glutathione depletion in adolescents with poorly controlled type 1 diabetes
postnatal diet-induced obesity in rats upregulates systemic and adipose tissue glucocorticoid metabolism during development and in adulthood
the effect of vitamin e on endothelial function of micro- and macrocirculation and left ventricular function in type 1 and type 2 diabetic patients
nitrergic neurodegeneration in cerebral arteries of streptozotocin-induced diabetic rats
a-series gangliosides mediate the effects of advanced glycation end products on pericyte and mesangial cell proliferation
differential effects of glucose and lactate on glucosensing neurons in the ventromedial hypothalamic nucleus
altered transcapillary escape of albumin and microalbuminuria reflects two different pathogenetic mechanisms
aldose reductase inhibition counteracts oxidative-nitrosative stress and poly(adp-ribose) polymerase activation in tissue sites for diabetes complications
decreased expression of pigment epithelium-derived factor is involved in the pathogenesis of diabetic nephropathy
development of a long-acting insulin analog using albumin fusion technology
genetic linkage and association of the growth hormone secretagogue receptor (ghrelin receptor) gene in human obesity
linkage of plasma adiponectin levels to 3q27 explained by association with variation in the apm1 gene
heritability of insulin secretion, peripheral and hepatic insulin action, and intracellular glucose partitioning in young and old danish twins
common polymorphisms in the adiponectin gene acdc are not associated with diabetes in pima indians
a genome scan for fasting insulin and fasting glucose identifies a quantitative trait locus on chromosome 17p
hiv-1 accessory protein vpr inhibits the effect of insulin on the foxo subfamily of forkhead transcription factors by interfering with their binding to 14-3-3 prote..
increased glucocorticoid receptor and 11-hydroxysteroid dehydrogenase type 1 expression in hepatocytes may contribute to the phenotype of type 2 diabetes in db/db m..
increased phosphorylation of akt substrate of 160 kda (as160) in rat skeletal muscle in response to insulin or contractile activity
growth hormone overexpression in the central nervous system results in hyperphagia-induced obesity associated with insulin resistance and dyslipidemia
enhanced hypothalamic amp-activated protein kinase activity contributes to hyperphagia in diabetic rats
islet-infiltrating b-cells in nonobese diabetic mice predominantly target nervous system elements
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