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《实验医学杂志》2006年第01期

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regulation of the germinal center gene program by interferon (ifn) regulatory factor 8/ifn consensus sequence-binding protein
histone deacetylase 2
purified hematopoietic stem cell engraftment of rare niches corrects severe lymphoid deficiencies without host conditioning
g-csf/scf reduces inducible arrhythmias in the infarcted heart potentially via increased connexin43 expression and arteriogenesis
disparate roles of atr and atm in immunoglobulin class switch recombination and somatic hypermutation
lat-mediated signaling in cd4+cd25+ regulatory t cell development
map kinase phosphatase 1 controls innate immune responses and suppresses endotoxic shock
identification of the target self-antigens in reperfusion injury
functional disruption of 4 integrin mobilizes bone marrow
cytokine signal transduction is suppressed in preselection double-positive thymocytes and restored by positive selection
uptake of leishmania major by dendritic cells is mediated by fc receptors and facilitates acquisition of protective immunity
dual specificity phosphatase 1 (dusp1) regulates a subset of lps-induced genes and protects mice from lethal endotoxin shock
role of mitochondrial glucocorticoid receptor in glucocorticoid-induced apoptosis
nod1 acts as an intracellular receptor to stimulate chemokine production and neutrophil recruitment in vivo
aid-dependent histone acetylation is detected in immunoglobulin s regions
activation of the flt3 signal transduction cascade rescues and enhances type i interferon
trail-expressing t cells induce apoptosis of vascular smooth muscle cells in the atherosclerotic plaque
innate secretory antibodies protect against natural salmonella typhimurium infection
the fate of human langerhans cells in hematopoietic stem cell transplantation
ccr5 deficiency increases risk of symptomatic west nile virus infection
induction of apobec3 family proteins, a defensive maneuver underlying interferon-induced anti
stoichiometry of the murine t cell receptor
an essential role for complement c5a in the pathogenesis of septic cardiac dysfunction
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