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《实验医学杂志》2003年第03期

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a mouse with a loss-of-function mutation in the c-cbl tkb domain shows perturbed thymocyte signaling without enhancing the activity of the zap-70 tyrosine kinase
a role of suppressor of cytokine signaling 3 (socs3/cis3/ssi3) in cd28-mediated interleukin 2 production
activation of dendritic cells through the interleukin 1 receptor 1 is critical for the induction of autoimmune myocarditis
changes in chromatin accessibility across the gm-csf promoter upon t cell activation are dependent on nuclear factor b proteins
transgenic expression of the activating natural killer receptor ly49h confers resistance to cytomegalovirus in genetically susceptible mice
the poxvirus protein a52r targets toll-like receptor signaling complexes to suppress host defense
t cell regulation as a side effect of homeostasis and competition
sustained activation of cell adhesion is a differentially regulated process in b lymphopoiesis
selective activation of the c-jun nh2-terminal protein kinase signaling pathway by stimulatory kir in the absence of karap/dap12 in cd4+ t cells
second class minorsmolecular identification of the autosomal h46 histocompatibility locus as a peptide presented by major histocompatibility complex class ii molecu..
restricting zap70 expression to cd4+cd8+ thymocytes reveals a t cell receptordependent proofreading mechanism controlling the completion of positive selection
regulatory t cells selectively express toll-like receptors and are activated by lipopolysaccharide
regulation of lymphocyte apoptosis by interferon regulatory factor 4 (irf-4)
prevention of arthritis by interleukin 10producing b cells
interleukin 1 receptor dependence of serum transferred arthritis can be circumvented by toll-like receptor 4 signaling
integrin-dependence of lymphocyte entry into the splenic white pulp
in vitro evidence that cytokine receptor signals are required for differentiation of double positive thymocytes into functionally mature cd8+ t cells
immunotherapy of cytotoxic t cellresistant tumors by t helper 2 cellsan eotaxin and stat6-dependent process
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