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《实验药学杂志》2005年第06期

two strikes lead to leukemia
defenseless against the common cold
too old to help
clean living staves off autoimmunity
signaling for nkt cell development : the sap
t cell aging : naive but not young
identifying genes that regulate bone remodeling as potential therapeutic targets
newly generated cd4 t cells in aged animals do not exhibit age-related defects in response to antigen
granulocyte/macrophage colony-stimulating factor and accessory cells modulate radioprotection by purified hematopoietic cells
regulation of anaphylactic responses by phosphatidylinositol phosphate kinase type i
platelet activation leads to activation and propagation of the complement system
a mutation in the icsbp1 gene causes susceptibility to infection and a chronic myeloid leukemia
transmission and accumulation of ctl escape variants drive negative associations between hiv polymorphisms and hla
cd44 is a determinant of inflammatory bone loss
interleukin-1 receptor-associated kinase-1 plays an essential role for toll-like receptor (tlr)7- and tlr9-mediated interferon- induction
inhibition of platelet-derived growth factor signaling attenuates pulmonary fibrosis
asthmatic bronchial epithelial cells have a deficient innate immune response to infection with rhinovirus
a role for fungal ?-glucans and their receptor dectin-1 in the induction of autoimmune arthritis in genetically susceptible mice
the nucleocytoplasmic shuttling protein ciz reduces adult bone mass by inhibiting bone morphogenetic protein
interleukin-7 is necessary to maintain the b cell potential in common lymphoid progenitors
in vivo depletion of lung cd11c+ dendritic cells during allergen challenge abrogates the characteristic features of asthma
complement receptors regulate differentiation of bone marrow plasma cell precursors expressing transcription factors blimp-1 and xbp-1
vaccinia virus protein a46r targets multiple toll-like
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