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《美国病理学杂志》2003年第03期

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a mouse with a loss-of-function mutation in the c-cbl tkb domain shows perturbed thymocyte signaling without enhancing the activity of the zap-70 tyrosine kinase
activation of dendritic cells through the interleukin 1 receptor 1 is critical for the induction of autoimmune myocarditis
changes in chromatin accessibility across the gm-csf promoter upon t cell activation are dependent on nuclear factor b proteins
dynamic changes during the immune response in t cellantigen-presenting cell clusters isolated from lymph nodes
extrathymic t cell lymphopoiesis
gata-1 as a regulator of mast cell differentiation revealed by the phenotype of the gata-1low mouse mutant
g-csfstimulated neutrophils are a prominent source of functional blys
immunotherapy of cytotoxic t cellresistant tumors by t helper 2 cells
in vitro evidence that cytokine receptor signals are required for differentiation of double positive thymocytes into functionally mature cd8+ t cells
integrin-dependence of lymphocyte entry into the splenic white pulp
interleukin 1 receptor dependence of serum transferred arthritis can be circumvented by toll-like receptor 4 signaling
low penetrance broad resistance and favorable outcome of interleukin 12 receptor ß1 deficiency
prevention of arthritis by interleukin 10producing b cells
regulatory t cells selectively express toll-like receptors and are activated by lipopolysaccharide
restricting zap70 expression to cd4+cd8+ thymocytes reveals a t cell receptordependent proofreading mechanism controlling the completion of positive selection
second class minors
selective activation of the c-jun nh2-terminal protein kinase signaling pathway by stimulatory kir in the absence of karap/dap12 in cd4+ t cells
sustained activation of cell adhesion is a differentially regulated process in b lymphopoiesis
t cell regulation as a side effect of homeostasis and competition
the poxvirus protein a52r targets toll-like receptor signaling complexes to suppress host defense
transgenic expression of the activating natural killer receptor ly49h confers resistance to cytomegalovirus in genetically susceptible mice
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