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《临床调查学报》2006年第03期

going nuclear in metabolic and cardiovascular disease
sorting out the roles of ppar in energy metabolism and vascular homeostasis
ppar and human metabolic disease
ppar: a dagger in the heart of the metabolic syndrome
liver x receptors as integrators of metabolic and inflammatory signaling
involvement of ppar nuclear receptors in tissue injury and wound repair
pgc-1 coactivators: inducible regulators of energy metabolism in health and disease
dichotomy of ca2+ in the heart: contraction versus intracellular signaling
a unique role for stat5 in recovery from acute anemia
an unexpected role for the anaphylatoxin c5a receptor in allergic sensitization
good news in the nuclear envelope: loss of lamin a might be a gain
going in circles: conserved mechanisms control radial patterning in the urinary and digestive tracts
ghrelin receptor mutations ― too little height and too much hunger
pericytes limit tumor cell metastasis
a homing mechanism for bone marrow
tbx18 regulates the development of the ureteral mesenchyme
local insp3-dependent perinuclear ca2+ signaling in cardiac myocyte excitation-transcription coupling
signals for stress erythropoiesis are integrated via an erythropoietin receptor
critical role of endothelial cxcr2 in lps-induced neutrophil migration into the lung
loss of irf-4
blocking the 4 integrinpaxillin interaction selectively impairs mononuclear leukocyte recruitment to an inflammatory site
similarities and differences between selective and nonselective baff blockade in murine sle
the latency-associated nuclear antigen of kaposi sarcoma
prelamin a and lamin a appear to be dispensable in the nuclear lamina
elastin fragments drive disease progression in a murine model of emphysema
role of the forkhead protein foxo1 in ? cell compensation to insulin resistance
platelet-derived or soluble cd154 induces vascularized allograft rejection independent of cell-bound cd154
a regulatory role for the c5a anaphylatoxin in type 2 immunity in asthma
cftr is required for pka-regulated atp sensitivity of kir1.1 potassium channels in mouse kidney
heme oxygenase-1 is a modulator of inflammation and vaso-occlusion in transgenic sickle mice
reversal of diet-induced hepatic steatosis and hepatic insulin resistance by antisense oligonucleotide inhibitors of acetyl-coa carboxylases 1 and 2
cholinergic dysfunction in a mouse model of alzheimer disease is reversed by an anti-a antibody
igg-blocking antibodies inhibit ige-mediated anaphylaxis in vivo through both antigen interception and fcriib cross-linking
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