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《动脉硬化血栓血管生物学》
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《动脉硬化血栓血管生物学》2007年第07期

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loss-of-function deletion of the steroid receptor coactivator-1 gene in mice reduces estrogen effect on the vascular injury response
cooperation of src-1 and p300 with nf- b and creb in angiotensin ii-induced il-6 expression in vascular smooth muscle cells
acyclic retinoid inhibits neointima formation through retinoic acid receptor beta-induced apoptosis
cyp26 inhibitor r115866 increases retinoid signaling in intimal smooth muscle cells
inducible camp early repressor inhibits growth of vascular smooth muscle cell
histamine upregulates the expression of inducible nitric oxide synthase in human intimal smooth muscle cells via histamine h1 receptor and nf- b signaling pathway
venous identity is lost but arterial identity is not gained during vein graft adaptation
endothelial outgrowth cells are not derived from cd133 + cells or cd45 + hematopoietic precursors
removal of fkbp12/12.6 from endothelial ryanodine receptors leads to an intracellular calcium leak and endothelial dysfunction
vascular endothelial growth factor is induced by the inflammatory cytokines interleukin-6 and oncostatin m in human adipose tissue in vitro and in murine adipose ti..
elevated cholesterol levels in the plasma membranes of macrophages inhibit migration by disrupting rhoa regulation
abca1-induced cell surface binding sites for apoa-i
apolipoprotein e interrupts interleukin-1ß signaling in vascular smooth muscle cells
expression of human osbp-related protein 1l in macrophages enhances atherosclerotic lesion development in ldl receptor-deficient mice
enhanced immune system activation and arterial inflammation accelerates atherosclerosis in lupus-prone mice
a specific role for enos-derived reactive oxygen species in atherosclerosis progression
type a eagerness-energy across developmental periods predicts adulthood carotid intima-media thickness
impact of serum amyloid a on tissue factor and tissue factor pathway inhibitor expression and activity in endothelial cells
endothelial fibrinolytic capacity predicts future adverse cardiovascular events in patients with coronary heart disease
activation of human platelets by misfolded proteins
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