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《动脉硬化血栓血管生物学》
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《动脉硬化血栓血管生物学》2004年第10期

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estrogen and hdl
crp and risk of coronary heart disease
molecular phenotypes of atherosclerosis
vascular cross-talk: a conversation
hepatic lipase, lipoprotein metabolism, and atherogenesis
regulation of plasma high-density lipoprotein levels by the abca1 transporter and the emerging role of high-density lipoprotein in the treatment of cardiovascular d..
endothelium-targeted gene and cell-based therapies for cardiovascular disease
finding vulnerable atherosclerotic plaques
leukotriene b4 strongly increases monocyte chemoattractant protein-1 in human monocytes
proinflammatory cytokines regulate lox-1 expression in vascular smooth muscle cells
pecam-1 interacts with nitric oxide synthase in human endothelial cells
expression of angiopoietin-2 in endothelial cells is controlled by positive and negative regulatory promoter elements
adp receptor p2y12 is expressed in vascular smooth muscle cells and stimulates contraction in human blood vessels
endogenous nitric oxide synthesis inhibitor asymmetric dimethyl l-arginine accelerates endothelial cell senescence
oxidized low-density lipoproteins stimulate extracellular matrix metalloproteinase inducer (emmprin) release by coronary smooth muscle cells
redd2 gene is upregulated by modified ldl or hypoxia and mediates human macrophage cell death
in vivo transcriptional response of cardiac endothelium to lipopolysaccharide
inhibition of rho-kinase leads to rapid activation of phosphatidylinositol 3-kinase/protein kinase akt and cardiovascular protection
carbon monoxide protects against cardiac ischemia―reperfusion injury in vivo via mapk and akt―enos pathways
cortical microvascular remodeling in the stenotic kidney
exogenous nadph increases cerebral blood flow through nadph oxidase
contrasting effects of oral versus transdermal estrogen on serum amyloid a (saa) and high-density lipoprotein
c-reactive protein-induced in vitro vasorelaxation is an artefact caused by the presence of sodium azide in commercial preparations
can exercise training with weight loss lower serum c-reactive protein levels?
c-reactive protein genotypes affect baseline, but not exercise training
mast cells in neovascularized human coronary plaques store and secrete basic fibroblast growth factor, a potent angiogenic mediator
antimonocyte chemoattractant protein-1 gene therapy attenuates graft vasculopathy
blockade of keratinocyte-derived chemokine inhibits endothelial recovery and enhances plaque formation after arterial injury in apoe-deficient mice
high-density lipoproteins retard the progression of atherosclerosis and favorably remodel lesions without suppressing indices of inflammation or oxidation
inhibition of cholesteryl ester transfer protein activity by jtt-705 increases apolipoprotein e
hemodynamic regulation of cd34+ cell localization and differentiation in experimental aneurysms
gene expression phenotypes of atherosclerosis
hyplip2, a new gene for combined hyperlipidemia and increased atherosclerosis
quantitative trait loci for apolipoprotein b, cholesterol, and triglycerides in familial combined hyperlipidemia pedigrees
low-density lipoprotein particle size loci in familial combined hyperlipidemia
segment-specific effects of cardiovascular risk factors on carotid artery intima-medial thickness in women at midlife
c-reactive protein, fibrin d-dimer, and risk of ischemic heart disease
pi3k-akt pathway suppresses coagulation and inflammation in endotoxemic mice
serum lipid levels and the risk of venous thrombosis
correction for villablanca et al., arterioscler thromb vasc biol 24 (6) 1055-1061.
clinical significance of coronary calcification
aortic stiffness does not mediate the relation between pulse pressure and crp
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