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《动脉硬化血栓血管生物学》
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《动脉硬化血栓血管生物学》2004年第07期

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gardner craddock mcmillan
myeloperoxidase and plaque vulnerability
origin of neointimal cells in autologous vein graft
intracellular cholesterol transport
vascular calcification
should progestins be blamed for the failure of hormone replacement therapy to reduce cardiovascular events in randomized controlled trials?
murine model of neointimal formation and stenosis in vein grafts
cyclophilin a is a proinflammatory cytokine that activates endothelial cells
deciphering regulatory patterns of inflammatory gene expression from interleukin-1―stimulated human endothelial cells
angiotensin ii stimulates the release of interleukin-6 and interleukin-8 from cultured human adipocytes by activation of nf-b
r-cadherin:?-catenin complex and its association with vascular smooth muscle cell proliferation
ccl11 (eotaxin) induces ccr3-dependent smooth muscle cell migration
microtubules regulate angiotensin ii type 1 receptor and rac1 localization in caveolae/lipid rafts
basic fibroblast growth factor
aif-1 expression modulates proliferation of human vascular smooth muscle cells by autocrine expression of g-csf
dc electric fields induce distinct preangiogenic responses in microvascular and macrovascular cells
enhanced p2x7 activity in human fibroblasts from diabetic patients
decreased number and impaired angiogenic function of endothelial progenitor cells in patients with chronic renal failure
muscarinic (m) receptors in coronary circulation
supplemented -tocopherol apparently does not enter the plaque compartment
balance between pgd synthase and pge synthase is a major determinant of atherosclerotic plaque instability in humans
morphologic findings of coronary atherosclerotic plaques in diabetics
progression of coronary artery calcium and risk of first myocardial infarction in patients receiving cholesterol-lowering therapy
infrarenal aortic diameter predicts all-cause mortality
atherosclerotic plaque smooth muscle cells have a distinct phenotype
human smooth muscle cell subpopulations differentially accumulate cholesteryl ester when exposed to native and oxidized lipoproteins
analysis of apolipoprotein a5, c3, and plasma triglyceride concentrations in genetically engineered mice
effect of pravastatin on low-density lipoprotein oxidation and myocardial perfusion in young adults with type 1 diabetes
hypochlorous acid, a macrophage product, induces endothelial apoptosis and tissue factor expression
small gtp-binding protein ral is involved in camp-mediated release of von willebrand factor from endothelial cells
protein c levels are regulated by a quantitative trait locus on chromosome 16
correction
vitamin e is not deficient in human atherosclerotic plaques
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